Help For Hearing Loss Sufferers

Hearing Sense Recovery

This product was created by Mark after he had gone through an experienceof getting his wife's hearing back. He was contacted by one of the most brilliant researchers on the topic of getting one's hearing back and he was able to conduct a research with him in the African tribes in Kenya, the superfoods they have used have made them become the country with the least hearing loss in the world. They have made an experiment on Mark's wife who was able to get her hearing back in merely three weeks. After that, Mark has decided to put the information out to the world so they can help their hearing and the hearing of their loved ones to finally experience life as it truly should. He put the information into an easy step by step guide that walks you through every single phase you have to go through to improve the hearing. The method is very easy to attain and it does not need any technical knowledge, you can easily get this product and you will be able to get your hearing back in just three weeks. The method that is used is also natural and safe and it definitely works no matter how bad the hearing condition is, it will heal and strengthen the cells responsible for hearing. Read more...

Hearing Sense Recovery Summary


4.7 stars out of 12 votes

Contents: Ebook
Author: Mark Allen
Price: $39.00

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My Hearing Sense Recovery Review

Highly Recommended

I usually find books written on this category hard to understand and full of jargon. But the author was capable of presenting advanced techniques in an extremely easy to understand language.

Overall my first impression of this book is good. I think it was sincerely written and looks to be very helpful.

Improve Your Hearing Naturally

What You'll Find Inside Improve Your Hearing Naturally: What Supplements Vitamins and Herbs You Need To Be Taking Right Now To Stop Hearing Loss, Restore Your Hearing and Open Up Those Muffled Ears . In Fact If You Missing These Nutrients In Your Diet Your Hearing Will Continue To Decline. How To Unblock And Clean Out Years Of Built Up Toxins From Your Ears and Ear Canals Instantly Improving Your Hearing! No More Muffled Hearing, How Would You Like To Hear Out Of Both Ears Equally? - Most People Are Completely Shocked When They See What Comes Out Of Their Ear Canals. Learn The Best Natural Home Remedies For Ear Infections, Ear Blockage, Ear Aches, Ringing In The Ears (Tinnitus) And Other Ear Problems - Plus How To Properly Clean Your Ears, Safely With No Risk Of Damage. Discover What Common Drugs Every Home Has In Their Medicine Cabinet You May Be Taking That Can Permanently Damage Your Hearing - Doctors Don't Tell You This And You Need To STOP Immediately

Improve Your Hearing Naturally Summary

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Organic acidurias and aminoacidurias

Lactic acidosis is present in blood and urine in Leigh's syndrome. This is usually due to X-linked or autosomal recessive nuclear gene mutations of one of the mitochon-drial respiratory chain complex I or IV enzymes or pyruvate dehydrogenase (X-linked PDHA1 gene). About 30 are due to mutations in mitochondrial DNA, which have been identified in at least 11 mitochondrial genes. The diagnostic features are a subacute encephalopathy, seizures (myoclonic or tonic-clonic), and progressive dementia with cerebellar and brainstem signs. Motor abnormalities include hypotonia, spasticity, ataxia, involuntary movements and bulbar problems. Vomiting, hyperventilation and abnormalities of thermoregulation are common. Optic atrophy, pigmentary retinopathy, deafness and cardiomyopathy are sometimes present. On imaging, basal ganglia lucencies are highly characteristic, and proton magnetic resonance spectro

Peroxisomal disorders

At least 17 autosomal recessive disorders are described due to abnormalities of 11 genes coding for peroxisomal enzymes. These enzymes act as metabolic pathways in the oxidation of long-chain fatty acids, necessary for myelin production. Zellweger syndrome and neonatal adrenoleuco-dystrophy are examples, presenting as severe seizure disorders often starting in the neonatal period and including all seizure types including infantile spasm. Other features include poor feeding, severe intellectual regression, bony stippling (chondrodysplasia punctata), retinal dystrophy, hearing loss, hypotonia and dysmorphic features (including high palate, high forehead and shallow orbital ridges). The EEG is severely abnormal. In some cases polymicrogyria is present. The diagnosis is made by measuring serum long-chain fatty acids in plasma, and then fibroblast culture. Mutations in 11 different PEX genes those that encode peroxins have been identified. Mutations of the PEX1 is the most common cause,...

Other Rare Epilepsies Associated with Simple Mendelian Inheritance

Several other less common epilepsies result from simple Mendelian inheritance, such as the progressive myoclonus epilepsies. However, these disorders are exceedingly rare and generally require diagnostic technologies not available in developing world settings. Mitochondrial disorders include myoclonic epilepsy with ragged red fibers (MERRF) and mitochondrial encephalomy-opathy with lactic acidosis and stroke-like episodes (MELAS). The overlapping features of mitochondrial disorders include seizures, dementia, progressive external ophthalmo-plegia, sensory neural hearing loss, cardiac abnormalities, and elevated levels of lactate and pyruvate.

Diagnostic approach

Based on the clinical presentation and progression of cognitive loss, associated symptoms, and ethnic origin, most of the other progressive myoclonic epilepsies can be potentially diagnosed. Other than the canonical features of myoclonus, generalized seizures, ataxia, and ragged red fibers in muscle, there are frequent other clinical abnormalities noted in MERRF, including sensorineural hearing loss, peripheral neuropathy, short stature, exercise intolerance, and optic atrophy. Less frequent clinical signs reported are cardiomyopathy, preexcitation arrhythmia (Wolf-Parkinson-White), pigmentary retinopathy, ophthalmoparesis, pyramidal signs, and multiple lipomas

Landau Kleffner Syndrome

The syndrome of acquired aphasia with convulsive disorder in children was first described by Landau and Kleffner32 in six children with normal early language development who suddenly developed aphasia in relation to an epileptic disorder. The language disturbance typically first involves verbal comprehension (classicly a verbal auditory agnosia) that may be mistaken for an acquired deafness. The usually gradual deterioration in verbal production follows the loss of comprehension, occurs together with it, or even precedes it. Several variables can influence the severity and the duration of the language disorder frequency of the epileptic discharges in the language zones, the duration of the epileptic disorder, the spread of the epileptic discharges to the homologous contralateral cortex, and the efficacy of the AED therapy.9394

Myoclonus Epilepsy With Ragged Red Fibers

Myoclonus epilepsy with ragged red fibers was first described in cases with a florid clinical myopathy and myoclonus epilepsy (22, 23). It is now clear that the clinical spectrum of MERRF is extremely broad. It should be suspected in a wide variety of situations, even when clinical and pathologic evidence of myopathy are absent (24). Symptoms may begin at any age, and there may be marked intrafamily variation in the age of onset and clinical severity (24, 25). The clinical features include myoclonus, tonic-clonic seizures, dementia, and ataxia, with less common findings of myopathy, neuropathy, deafness, and optic atrophy. Some cases show striking axial lipomas. Occasional patients or families have focal neurologic events, and there is an overlap with the syndrome of mitochondrial encephalomyopathy, lactic acido-sis, and strokelike episodes (MELAS), in which strokelike Diagnosis can usually be suspected clinically but may be difficult to confirm with laboratory markers. The clinical...

Diagnosing The Specific Type Of

Although patients with the PME syndrome superficially may appear to have similar clinical features, knowledge of the specific clinical patterns of the common causes of PME often allows the differential diagnosis to be narrowed. Age at onset of symptoms provides some guidance in making the diagnosis, although MERRF may begin at any age. Certain seizure patterns are helpful very prominent myoclonus suggests Unverricht-Lundborg disease, MERRF, or sialidosis. Partial seizures, particularly of occipital origin, can occur in a variety of the disorders but are often noted in Lafora disease. Characteristic fun-dal changes are almost invariable in sialidosis and are frequent in the NCLs. Dementia is a constant feature of Lafora disease and NCLs, whereas it is characteristically absent or mild in Unverricht-Lundborg disease and sialidosis type I. The presence of deafness, lipomas, optic atrophy, myopathy, or neuropathy are clinical pointers to MERRF. Neuropathy may also occur in sialidosis....

Clinical Manifestations

The onset of LKS usually occurs in children older than 4 years (26), with a range of 3 to 10 years (27). LKS may first manifest as an apparent word deafness or a verbal auditory agnosia. Seizures and behavior disturbances, particularly attention deficits and hyperactivity, each occur in approximately two-thirds of children with LKS (5). The majority of cases are classified as idiopathic, although any pathologic process affecting the auditory cortex may cause LKS. Symptomatic cases have been described (see the section on differential diagnosis), and we have seen symptomatic LKS caused by a left temporal oligodendroglioma, with clinical improvement noted after resection. The classical features of LKS are a verbal auditory agnosia (word deafness) followed by language regression, seizures, or both in a previously normal child who has an epileptiform EEG. An important corollary is normal hearing, because a central disorder cannot be diagnosed in the presence of peripheral dysfunction....

Temperal Flat Cake Like Structure Found In The Uterus During Pregnancy

Malformations in the fetus. A birth defect may affect how the body looks, function, or both. It may be found before birth, at birth, or anytime after birth. Most defects are discovered within the first year of life. Some birth defects (cleft lip or clubfoot) are easy to see, but others (heart defects or hearing loss) are found using special tests (X-rays, CAT scans, echocardiography, or hearing tests). Birth defects can vary from mild to severe some may cause the baby to die. Babies with birth defects may need surgery or other medical treatments, but with medical care they usually lead normal lives.

Mitochondrial cytopathy myoclonic epilepsy with ragged red fibres [MERRF

The mitochondrial cytopathy that typically causes progressive myoclonic epilepsy is the syndrome of myoclonus epilepsy with ragged red fibres (MERRF). This is a multisystem disorder with a very variable phenotype, in which myoclonic seizures are often the first symptom, followed by generalized epilepsy, myopathy, ataxia and dementia. Other features are short stature, deafness, optic atrophy, retinopathy, ophthalmoparesis and cardiomyopathy with

Long QT Disorders

Difficulty in the diagnosis of the syndrome of Jervell and Lange-Nielsen, in which congenital deafness is associated with an autosomal recessive inheritance.23 Much more difficult is the Romano-Ward syndrome, which is dominantly inherited but with incomplete penetrance. There is a degree of overlap between the stimuli which induce the neurally mediated syncopes and those which trigger the ventricular tachyarrhythmias of the long QT syndrome. The two situations in which cardiac arrhythmias have to be actively considered are those in which convulsions occur

Prenatal Infections

Prenatal cytomegalovirus (CMV) infection occurs through the first to third trimesters in symptomatic patients. CMV has an affinity for the rapidly proliferating subependymal cells lining the ventricles. Viral multiplication and subsequent calcium deposition result in brain disruption or dysgenesis with periventricular or diffuse calcification. Microcephaly (50 ), hydrocephalus, neuronal migration disorders, porencephaly, and polycystic encephalomalasia are observed. Mental retardation, visual disturbance, hearing loss, language disorders, and epilepsy can be observed.23,29,30


Juvenile sialidosis type II presents as a PME with features similar to those of sialidosis type I except that onset is sometimes a little later. There may be additional features of coarse facies, corneal clouding, dysostosis multiplex, hearing loss, and low intellect, which may be present from early life (59, 60).

Metabolic Disorders

Skin rashes and striking neurologic symptoms with seizures are prominent signs of biotinidase deficiency. Hypotonia, developmental delay, and seizures are presenting features in the neonatal form. In the late-onset type, 1 week to 2 years onset, the most common presenting feature is seizures. The most common seizure type is myoclonus, although generalized tonic-clonic and focal clonic seizures have been described (61). Ataxia and hypotonia are present, as are rash and alopecia. Hearing loss may occur. Treatment is oral biotin with rapid response within 24 hours (61, 62).

Hearing Aids Inside Out

Hearing Aids Inside Out

Have you recently experienced hearing loss? Most probably you need hearing aids, but don't know much about them. To learn everything you need to know about hearing aids, read the eBook, Hearing Aids Inside Out. The book comprises 113 pages of excellent content utterly free of technical jargon, written in simple language, and in a flowing style that can easily be read and understood by all.

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